The shrine of knowledge.


excitotoxicity. exaggerated and continuous stimulation by a neurotransmitter, especially in those neuronal systems which use glutamate as the transmitter.

Excitotoxicity is described in multiple online sources, as addition to our editors' articles, see section below for printable documents, Excitotoxicity books and related discussion.

Suggested Pdf Resources

Chronic Microglial Activation and Excitotoxicity Secondary to
damaged by neuroinflammation and attendant excitotoxicity. 1. 2.
374 Blaylock Nov 16
production, also magnify excitotoxic sensitivity of neurons and their elements.
Review Article: The Central Role of Excitotoxicity in
Named in 1969 by Dr.
PDZ Protein Interactions Underlying NMDA Receptor- Mediated
excitotoxicity without inhibiting NMDAR ionic currents and cal- cium signaling ( Sattler et interactions responsible for excitotoxicity in cortical neuronal cultures.
Excitotoxicity, Synaptic Repair, and Functional Recovery in the

Suggested News Resources

What the spellers say...
When I stepped up to the microphone, I was told to spell the word “excitotoxicity”. I was giddy with excitement because I knew that word very well. But it was a long word, so I rushed through the spelling.
Neurodegenerative disorders: Restoring the balance

Suggested Web Resources

Excitotoxicity - Wikipedia, the free encyclopedia
Excitotoxicity and cell damage
Excitotoxicity and the NMDA receptor
Excitotoxicity, a phenomenon that which was first described by Olney in the seventies, implies the activation in the CNS of the so-called glutamate receptors.
The role of excitotoxicity in neurodegenerative disease: implications
The role of excitotoxicity in neurodegenerative disease: implications for therapy. Doble A. Neuroscience Dept.
Mechanism of Glutamate Excitotoxicity - YouTube
Apr 25, 2010 This is a brief, simple animation of the mechanism for glutamate excitotoxicity following an ischemic stroke.

Great care has been taken to prepare the information on this page. Elements of the content come from factual and lexical knowledge databases, library and third-party sources. We appreciate your suggestions and comments on further improvements of the site.

Discussion Forum
Place for your opinion